Varicella-Zoster Virus Fails to Induce Immunoglobulin G Fc Receptors in Infected Human Cells

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Abstract
Varicella-zoster virus infection of human fibroblasts and human brain cells produces characteristic cytopathogenic effects and virus-specific antigens. Receptors for the Fc portion of IgG, which were noted on cells infected with other human herpesviruses, could not be detected either by hemadsorption of sensitized sheep erythrocytes or by binding or radiolabeled IgG. Herpes simplex virus-infected cells were positive under the same conditions.