CARBOHYDRATE METABOLISM

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Abstract
REVIEW OF THE LITERATURE Evidences of an abnormal carbohydrate metabolism, hyperglycemia, glycosuria and diminished tolerance, as judged by the dextrose tolerance curve, have been described in hyperthyroidism.1 The point has been at issue whether these findings could be explained as incidents of the accelerated metabolism or whether an intimate relationship between the pancreas and the thyroid, with the latter in the rôle of an antagonist, would not have to be invoked to explain the findings. John2 reviewed the conflicting literature in 1927, and stated: "A number of authors, with whom I agree, believe that hyperthyroidism plays a fundamental etiologic rôle in the disturbance of endocrine equilibrium which constitutes the diabetic syndrome." It is known3 that after the ingestion of thyroid substance the liver no longer retains glycogen as it previously did (increased glycogenolysis?). Du Bois4 assumed that this explains the fact that although his patients with

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