Mitochondrial functions during cell death, a complex (I–V) dilemma
- 2 May 2003
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 10 (5) , 488-492
- https://doi.org/10.1038/sj.cdd.4401225
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- Endonuclease G: a mitochondrial protein released in apoptosis and involved in caspase-independent DNA degradationCell Death & Differentiation, 2001
- The cytotoxic action of Bax on yeast cells does not require mitochondrial ADP/ATP carrier but may be related to its import to the mitochondriaFEBS Letters, 2000
- A Comparative Study of Apoptosis and Necrosis in HepG2 Cells: Oxidant-Induced Caspase Inactivation Leads to NecrosisBiochemical and Biophysical Research Communications, 1999
- Superoxide in ApoptosisJournal of Biological Chemistry, 1998
- Anti-apoptotic oncogenes prevent caspase-dependent and independent commitment for cell deathCell Death & Differentiation, 1998
- Commitment to cell death measured by loss of clonogenicity is separable from the appearance of apoptotic markersCell Death & Differentiation, 1998
- Mitochondrial control of apoptosisPublished by Elsevier ,1997
- Apoptotic features of selective neuronal death in ischemia, epilepsy and gpI20 toxicityTrends in Neurosciences, 1996
- Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death.The Journal of Experimental Medicine, 1995
- Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.The Journal of Experimental Medicine, 1995