Minimal BH3 Peptides Promote Cell Death by Antagonizing Anti-apoptotic Proteins
Open Access
- 1 May 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (21) , 19426-19435
- https://doi.org/10.1074/jbc.m209472200
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Direct addition of BimL to mitochondria does not lead to cytochrome c releaseFEBS Letters, 2002
- The channel of deathThe Journal of cell biology, 2001
- Discovery of Small-Molecule Inhibitors of Bcl-2 through Structure-Based Computer ScreeningJournal of Medicinal Chemistry, 2001
- A Molecular WhodunitScience, 2001
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- BID-dependent and BID-independent pathways for BAX insertion into mitochondriaCell Death & Differentiation, 2000
- Preservation of Mitochondrial Structure and Function after Bid- or Bax-Mediated Cytochrome c ReleaseThe Journal of cell biology, 2000
- Movement of Bax from the Cytosol to Mitochondria during ApoptosisThe Journal of cell biology, 1997
- Structure of Bcl-x L -Bak Peptide Complex: Recognition Between Regulators of ApoptosisScience, 1997
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993