Myocardial Function With Reduced Expression of the Sodium-Calcium Exchanger
- 30 September 2010
- journal article
- Published by Elsevier in Journal of Cardiac Failure
- Vol. 16 (9) , 786-796
- https://doi.org/10.1016/j.cardfail.2010.03.012
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Combined Deficiency of Dystrophin and β1 Integrin in the Cardiac Myocyte Causes Myocardial Dysfunction, Fibrosis and CalcificationCirculation Research, 2008
- Hypertrophy and Heart Failure in Mice Overexpressing the Cardiac Sodium-Calcium ExchangerJournal of Cardiac Failure, 2007
- Role of AT1 receptor in isoproterenol-induced cardiac hypertrophy and oxidative stress in miceJournal of Molecular and Cellular Cardiology, 2007
- Mechanism of shortened action potential duration in Na+-Ca2+ exchanger knockout miceAmerican Journal of Physiology-Cell Physiology, 2007
- Cardiac-Specific Ablation of the Na + -Ca 2+ Exchanger Confers Protection Against Ischemia/Reperfusion InjuryCirculation Research, 2005
- Genetic manipulation of cardiac Na+/Ca2+ exchange expressionBiochemical and Biophysical Research Communications, 2004
- Functional Adult Myocardium in the Absence of Na + -Ca 2+ ExchangeCirculation Research, 2004
- Forefront of Na+/Ca2+ Exchanger Studies: Role of Na+/Ca2+ Exchanger – Lessons From Knockout MiceJournal of Pharmacological Sciences, 2004
- Targeted Disruption of Na+/Ca2+ Exchanger Gene Leads to Cardiomyocyte Apoptosis and Defects in HeartbeatPublished by Elsevier ,2000
- Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human HeartsNew England Journal of Medicine, 1982