Toll-like Receptor 4 Is a Sensor for Autophagy Associated with Innate Immunity
Top Cited Papers
Open Access
- 1 July 2007
- Vol. 27 (1) , 135-144
- https://doi.org/10.1016/j.immuni.2007.05.022
Abstract
No abstract availableKeywords
Funding Information
- American Historical Association
- National Heart, Lung, and Blood Institute
- National Institute of Allergy and Infectious Diseases
- National Institutes of Health (1F32 HL078520-01)
- Alpha-1 Foundation
This publication has 31 references indexed in Scilit:
- Growth Factor Regulation of Autophagy and Cell Survival in the Absence of ApoptosisPublished by Elsevier ,2005
- Activation and signaling of the p38 MAP kinase pathwayCell Research, 2005
- Eating Oneself and Uninvited GuestsCell, 2005
- Autophagy Defends Cells Against Invading Group A StreptococcusScience, 2004
- Signal transduction by the lipopolysaccharide receptor, Toll‐like receptor‐4Immunology, 2004
- RIP1 is an essential mediator of Toll-like receptor 3–induced NF-κB activationNature Immunology, 2004
- Development by Self-DigestionDevelopmental Cell, 2004
- Disruption of the c-JUN-JNK Complex by a Cell-permeable Peptide Containing the c-JUN δ Domain Induces Apoptosis and Affects a Distinct Set of Interleukin-1-induced Inflammatory GenesJournal of Biological Chemistry, 2003
- Innate immune sensing and its roots: the story of endotoxinNature Reviews Immunology, 2003
- Inducible Nitric-oxide Synthase Is Regulated by the Proteasome Degradation PathwayJournal of Biological Chemistry, 2001