Small heat shock proteins differentially affect Aβ aggregation and toxicity
- 1 August 2006
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 347 (2) , 527-533
- https://doi.org/10.1016/j.bbrc.2006.06.128
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Anti-amyloidogenic activity of tannic acid and its activity to destabilize Alzheimer's β-amyloid fibrils in vitroBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2004
- Transglutaminase catalyzes differential crosslinking of small heat shock proteins and amyloid-βFEBS Letters, 2004
- Heat Shock Protein 70 Participates in the Neuroprotective Response to Intracellularly Expressed β-Amyloid in NeuronsJournal of Neuroscience, 2004
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of PathogenesisScience, 2003
- The green tea polyphenol (−)-epigallocatechin gallate attenuates β-amyloid-induced neurotoxicity in cultured hippocampal neuronsLife Sciences, 2001
- Interaction between β‐amyloid and lens αB‐crystallinFEBS Letters, 2000
- The Molecular Chaperone αB-crystallin Enhances Amyloid β NeurotoxicityBiochemical and Biophysical Research Communications, 1999
- A Strategy for Designing Inhibitors of β-Amyloid ToxicityJournal of Biological Chemistry, 1996
- Arrest of -Amyloid Fibril Formation by a Pentapeptide LigandJournal of Biological Chemistry, 1996
- Selective Inhibition of Aβ Fibril FormationJournal of Biological Chemistry, 1996