Subclinical Renal Response to Streptococcal Infection

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Abstract
INVESTIGATORS concerned with the pathogenesis of glomerulonephritis have studied streptococcal infection in man, and its consequences, for many years. Whereas the incidence of rheumatic fever after streptococcal infection has been said to be reasonably constant at approximately 3 per cent,1 the attack rate of acute glomerulonephritis is known to vary considerably. Among the factors considered significant in this variability may be cited the recognized differences in nephritogenic capacity of the many types of beta-hemolytic Group A streptococci, the definitive population under study, the possible influence of early antibiotic therapy and the as yet largely unexplored field of genetic predisposition to . . .