Type I Transforming Growth Factor β Receptor Binds to and Activates Phosphatidylinositol 3-Kinase
Open Access
- 1 March 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (11) , 10870-10876
- https://doi.org/10.1074/jbc.m413223200
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Smad-dependent and Smad-independent pathways in TGF-β family signallingNature, 2003
- The phosphatidylinositol 3-Kinase–AKT pathway in human cancerNature Reviews Cancer, 2002
- TGF-β signaling: positive and negative effects on tumorigenesisCurrent Opinion in Genetics & Development, 2002
- Transforming Growth Factor β Enhances Epithelial Cell Survival via Akt-dependent Regulation of FKHRL1Molecular Biology of the Cell, 2001
- Phosphatidylinositol 3-Kinase Function Is Required for Transforming Growth Factor β-mediated Epithelial to Mesenchymal Transition and Cell MigrationJournal of Biological Chemistry, 2000
- Signaling by Distinct Classes of Phosphoinositide 3-KinasesExperimental Cell Research, 1999
- Phosphatidylinositol 3-Kinase Is Required for Growth Factor–Induced Amino Acid Uptake by Vascular Smooth Muscle CellsArteriosclerosis, Thrombosis, and Vascular Biology, 1999
- TGF-β SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1998
- Transforming growth factor β-induced phosphorylation of Smad3 is required for growth inhibition and transcriptional induction in epithelial cellsProceedings of the National Academy of Sciences, 1997
- TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4The EMBO Journal, 1997