The fetal origin of adult diseases
- 1 January 2003
- journal article
- review article
- Published by Taylor & Francis in Journal of Obstetrics and Gynaecology
- Vol. 23 (5) , 555-560
- https://doi.org/10.1080/0144361031000156483
Abstract
In the last decade, the development of Barker's hypothesis of fetal programming opened the field for extensive research into the fetal origin of adult diseases. The association between low birth weight, which reflects intrauterine nutritional status, and the development of adult diseases has been confirmed in many studies for type 2 diabetes, hypertension and coronary heart diseases. Many other adulthood diseases were investigated for the same association with variable outcomes. These will be presented in this review.Keywords
This publication has 31 references indexed in Scilit:
- Early and late growth and blood pressure in adolescenceJournal of Epidemiology and Community Health, 2003
- Hepatoblastoma and low birth weight: A trend or chance observation?Medical and Pediatric Oncology, 2002
- Early growth and coronary heart disease in later life: longitudinal studyBMJ, 2001
- Catch-up growth in childhood and death from coronary heart disease: longitudinal studyBMJ, 1999
- Development of unfavorable hepatoblastoma in children of very low birth weightCancer, 1998
- Mother's weight in pregnancy and coronary heart disease in a cohort of finnish men: follow up studyBMJ, 1997
- ARTICLES: Testicular Nonseminoma and Seminoma in Relation to Perinatal CharacteristicsJNCI Journal of the National Cancer Institute, 1996
- Nutrition and the immune systemProceedings of the Nutrition Society, 1993
- Type 2 (non-insulin-dependent) diabetes mellitus: the thrifty phenotype hypothesisDiabetologia, 1992
- Fetal and infant growth and impaired glucose tolerance at age 64.BMJ, 1991