Hypoaldosteronism

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Abstract
MOST patients with Addison's disease can be demonstrated to have a defect in electrolyte regulation.1 The resulting tendency to sodium depletion and hyperpotassemia is often an important feature of the presenting clinical syndrome, but almost always there are associated signs and symptoms ascribable to a deficiency in the production of the glucocorticoid hormones, particularly hydrocortisone. Even when the signs of mineralocorticoid deficiency are prominent, spontaneous potassium intoxication is unusual, since the serum concentration of potassium rarely reaches levels capable of having serious cardiac effects.2 The purpose of the present report is to describe studies carried out on a patient with . . .