UNESTERIFIED FATTY ACID IN HUMAN BLOOD PLASMA. II. THE TRANSPORT FUNCTION OF UNESTERIFIED FATTY ACID

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Abstract
A method for the determination of UFA in blood plasma is described. Measurements of arteriovenous differences in humans demonstrated transport of UFA from depots to heart, liver, and probably skeletal muscle. Administration of glucose with insulin caused a cessation of transport, together with a decrease in the arterial concentration of UFA. Decreased venous UFA followed the administration of alanine, glutamic acid, and leucine. Adipose tissue probably maintains a "caloric homeostasis" by liberating UFA into the blood when exogenous non-fat calories are not available.